doi.org/10.3791/50873
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An Orthotopic Murine Model of Human Prostate Cancer Metastasis
Northwestern University. This orthotopic model of human prostate cancer allows for quantification of tumor size, circulating tumor cells, and formation of distinct metastasis to the lung. As cells must escape the primary organ, enter the blood stream, and implant into a secondary site, this model effectively recapitulates the scenario in humans.
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An Orthotopic Murine Model of Human Prostate Cancer Metastasis
Northwestern University. This orthotopic model of human prostate cancer allows for quantification of tumor size, circulating tumor cells, and formation of distinct metastasis to the lung. As cells must escape the primary organ, enter the blood stream, and implant into a secondary site, this model effectively recapitulates the scenario in humans.
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https://doi.org/10.3791/50873An Orthotopic Murine Model of Human Prostate Cancer Metastasis
Northwestern University. This orthotopic model of human prostate cancer allows for quantification of tumor size, circulating tumor cells, and formation of distinct metastasis to the lung. As cells must escape the primary organ, enter the blood stream, and implant into a secondary site, this model effectively recapitulates the scenario in humans.
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30- titleAn Orthotopic Murine Model of Human Prostate Cancer Metastasis
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- descriptionNorthwestern University. This orthotopic model of human prostate cancer allows for quantification of tumor size, circulating tumor cells, and formation of distinct metastasis to the lung. As cells must escape the primary organ, enter the blood stream, and implant into a secondary site, this model effectively recapitulates the scenario in humans.
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2- og:titleAn Orthotopic Murine Model of Human Prostate Cancer Metastasis
- og:descriptionNorthwestern University. This orthotopic model of human prostate cancer allows for quantification of tumor size, circulating tumor cells, and formation of distinct metastasis to the lung. As cells must escape the primary organ, enter the blood stream, and implant into a secondary site, this model effectively recapitulates the scenario in humans.
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117- http://www.ncbi.nlm.nih.gov/pubmed?term=((A+fluorescent+orthotopic+bone+metastasis+model+of+human+prostate+cancer%5BTitle%5D)+AND+%22Cancer+Res%22%5BJournal%5D)
- http://www.ncbi.nlm.nih.gov/pubmed?term=((Androgen-independent+prostate+cancer+progression+in+the+TRAMP+model%5BTitle%5D)+AND+%22Cancer+Res%22%5BJournal%5D)
- http://www.ncbi.nlm.nih.gov/pubmed?term=((Dietary+genistein+inhibits+metastasis+of+human+prostate+cancer+in+mice%5BTitle%5D)+AND+%22Cancer+Res%22%5BJournal%5D)
- http://www.ncbi.nlm.nih.gov/pubmed?term=((Endoglin+inhibits+prostate+cancer+motility+via+activation+of+the+ALK2-Smad1+pathway%5BTitle%5D)+AND+%22Oncogene%22%5BJournal%5D)
- http://www.ncbi.nlm.nih.gov/pubmed?term=((Endoglin+suppresses+human+prostate+cancer+metastasis%5BTitle%5D)+AND+%22Clin.+Exp.+Metastasis%22%5BJournal%5D)